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Intragastric gastric potentials were recorded from the dorsal vagal complex (DVC) in urethane-anesthetized cats with intact vagal nerves, and after vagotomy, vagal deafferentation, and sinoaortic denervation. Gastric potentials (measured as the difference between direct and gastric transmural potentials) were obtained in response to mechanical distension of the stomach by balloon inflation. Gastric potentials were present in animals without vagal nerves, but were absent in animals with vagotomy, vagal deaff
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Adult T-cell leukemia (ATL) and HTLV-1-associated myelopathy/tropical spastic paraparesis (HAM/TSP) belong to the category of “tropical spastic paraparesis,” and are chronic progressive neurological diseases with different pathogenic mechanisms. We herein reviewed HTLV-1 immunopathology and viral pathogenic mechanisms in ATL and HAM/TSP. In contrast to HAM/TSP, in ATL HTLV-1 can be eradicated by cytotoxic T cells (CTLs) and ATL cells can be eliminated from the circulation and bone marrow by CD8(+) CTLs, and ATL cells exhibit impaired expression of chemokine receptors and adhesion molecules and downregulation of adhesion molecule expression by dendritic cells (DCs), resulting in resistance to CTL-mediated lysis. Furthermore, ATL cells can evade CTL lysis via Fas-independent mechanisms, which involve Fas/Fas ligand (FasL) interaction, Fas-associated death domain (FADD)/Fas interaction, and Akt activation. In addition to these mechanisms, we found that several genes regulated by interferon-α (IFNα) can be upregulated in ATL cells. Notably, IFNα induces regulatory cell populations with apoptosis-suppressing activity in ATL cells, and we found that interleukin (IL) 10 secreted from ATL cells induced resistance to CTL-mediated apoptosis. Moreover, we found that ATL cells can produce a high level of prostaglandin E2 (PGE2), and PGE2 inhibited CTL function and induced apoptosis via EP2 and EP4 receptor-dependent pathways. Finally, we found that there was a close correlation between HAM/TSP and HTLV-1 carriers, and several chemokines and their receptors were also upregulated in HAM/TSP. Taken
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