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Neuronal toxicity of sodium arsenite in rats: enhancement of the protective effect by co-administration of vitamin E.
Neurotoxicity of arsenic (As) is a major health concern because of frequent exposure to As-contaminated drinking water. Food consumption is the major route of As exposure in humans. The present study was designed to assess the effect of the food on the toxicity of As. For this purpose, Wistar rats were treated with a low dose of sodium arsenite (NaAsO2, 1 mg/kg, ip) once a week for six weeks. We used several endpoints to evaluate the long-term effects of acute NaAsO2 exposure on the central nervous system (CNS). We focused on brain histopathology, lipid peroxidation, antioxidant defenses, and neurobehavioral alterations (body weight, biochemical assay, measurement of brain mitochondrial functions, and neurobehavioral tests). Administration of NaAsO2 led to a significant decrease in body weight associated with a significant increase in the brain lipid peroxidation, as well as a decrease in neuronal and glial cell densities, and alterations in some mitochondrial functions (respiratory control and NADH oxidation). NaAsO2-induced behavioral effects were clearly observed. Furthermore, NaAsO2 treatment resulted in anxiogenic behavior and caused learning and memory deficits. The neuroprotective effect of co-administration of the antioxidant vitamin E, a potent ROS scavenger, was clearly detected. These results suggest that the toxic effect of NaAsO2 may be potentiated when rats are fed a high fat diet. Vitamin E might be a suitable choice for the prevention of chronic low dose As exposure because it may act by a mechanism of neuroprotection.Dasht-e-Karak-e-Ghazi
Dasht-e-Karak-e-Ghazi (; also Romanized as Dasht-e-Karak-e-Ghāzī; also known as Dastkor Daraghazi) is a village in Cham Chamam Rural District, Chendar District, Savojbolagh County, Alborz Province, Iran. At the 2006 census, its population was 219
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