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The muscular dystrophy caused by the mutation of the protein-encoding gene for fukutin-related protein (FKRP) is characterized by high molecular weight aggregates of mutant FKRP in the cytoplasm. The direct defect of FKRP in muscle fibers was studied by immunohistochemical analysis using a human FKRP-specific antibody. FKRP immunoreactivity was highly concentrated at the Z-line in the normal mice, whereas the FKRP immunoreactivity was diffusely distributed in the normal mice. The FKRP immunoreactivity was observed in the sarcomere-containing portions of the dystrophic muscle fibers, and the abnormal immunoreactivity was also dispersed throughout the sarcoplasmic reticulum. Similar FKRP immunoreactivity was observed in the normal and in the dystrophic muscle fibers of the db/db mice. We have also examined the localization of sympathetic nerve fibers immunoreactive to tyrosine hydroxylase in the muscle, using double immunofluorescence. The tyrosine hydroxylase immunoreactivity was observed to be dispersed throughout the muscle fibers, similar to the FKRP immunoreactivity.Kur (surname)

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